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Serum autoantibodies to brain in Landau-Kleffner variant, autism, and other neurologic disorders

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(2) feedback inhibitory effects of GSH on glutamate cysteine ligase (GCL) activity (the first biosynthetic enzyme), which is regulated by the availability of cysteine (Jones, Direct delivery of GCC to the brain has been previously reported to protect against cellular depletion of GSH in the brain using the Buthionine sulfoximine (BSO) model of depletion and increase GSH levels in the pre-perturbed model (Pileblad and Magnusson, Previous studies such as those by Dringen et al

Acta Biomater 76:239256

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Thus, supplementation with glutathione could address this deficiency.* Although researchers have not yet conducted studies on glutathione for IBD in either animal models or in humans, there is evidence that other antioxidant therapies can address symptomspossibly by raising levels of glutathione